Prevention of polycystic kidney disease, resistance to polyc

Prevention of polycystic kidney disease, resistance to polyc

Renal cyst has no corresponding renal cyst etiology in TCM, kidney cyst etiology according to clinical manifestations attributable "hematuria", "pain", "accumulation", "consumptive disease" and other disease areas. "Lingshu. The dirty article pointed out that the kidney is good disease, low back pain, easy to damage to evil". The "pivot. Expanding on the article "and" records, such as "kidney swelling" kidney distention, abdominal fullness lead back central course, waist and thigh pain and described the main syndrome characteristics and renal cyst were similar.

Summarize the history of literature, combined with clinical characteristics, etiology of renal cyst experts, renal cyst etiology and kidney essence deficiency, poor blood, endogenous phlegm, context and resistance related to surgical treatment of simple temporary solution, can not increase fundamentally inhibiting cyst, so the best application of Integrated Chinese and Western medicine treatment. By promoting blood circulation, removing blood stasis removing effect, which naturally reduced renal cyst. The specific causes and processes of renal cysts are as follows:

1, simple renal cyst etiology

Simple renal cysts may be derived from the retention of cysts in the development of immature glomeruli, or as a result of inflammation and ischemia.

2, the incidence of polycystic kidney disease

Department of polycystic kidney, renal cortex, medulla is a genetic disorder of numerous follicles, when the cyst is small does not pose any threat to renal cyst, but with the age of the patient increases, increases in the process because of the oppression of the kidney and renal cyst within the blood vessels, causing kidney microcirculation disorder, make kidney ischemia, hypoxia, resulting in renal capillary endothelial cell damage, and attract the infiltration of inflammatory cells in the blood circulation, and increased glomerular topical inflammatory reaction, initiated renal fibrosis. If not timely and effective treatment to control the damaged tissue cells will release a series of induced renal toxicity factor, the phenotypic transformation of renal cells, accelerate the injury rate of renal function, especially in the distribution of renal interstitial fibroblasts into myofibroblasts, secretion lots is not easy by extracellular matrix degradation and immune complexes, because the dynamic imbalance of synthesis and degradation of extracellular matrix, extracellular matrix accumulation, resulting in glomerulosclerosis, tubular atrophy, renal fibrosis that gradually replaced the healthy kidney, kidney function gradually lost, the clinical development of end-stage renal. Patients with renal failure, namely uremia.

 

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