In the treatment of chronic kidney disease, when the kidney is damaged to renal failure, modern western medicine will largely recommend dialysis or renal transplantation. Dialysis is used as an alternative to renal replacement therapy in the kidney, and the patients who have entered the end-stage renal failure have been treated with dialysis at the same time, mostly hemodialysis. However, long-term dialysis will lead to a dependency that leads to complete loss of renal function.
While the kidney transplantation, renal transplantation for kidney rejection and the body is scarce and expensive surgery, disease and healing period of problem has plagued both doctors and patients, the general attitude of kidney transplant patients with renal failure in common is: the last choice.
The progression of renal failure to end-stage uremia, many people say, will worsen the condition of kidney disease due to poor control. In fact, this is only a relatively general statement, the real reason is renal fibrosis, the process of renal fibrosis if not blocked, it will lead to the loss of effective renal units and progressive decline of renal function. Therefore, uremia is the ultimate manifestation of enlargement of renal fibrosis. Therefore, the treatment of stable renal failure patients, prevention and development to hemodialysis stage, the key is: to block the progress of renal fibrosis, repair damaged kidney units, restore damaged kidney function.
Delaying or blocking the progression of renal function in early and intermediate chronic renal failure patients will reduce the incidence of uremia, relieve symptoms and improve the general state of the patient. There are many factors influencing the course of progressive chronic renal failure, in addition to different causes, but also affected by the glomerular filtration, high metabolism in renal tubules, metabolic acidosis, secondary hyperparathyroidism and metastatic calcification, hyperlipidemia, some other important factors of uremic toxins. It may also be related to the patient's special genetic genes, which, in the course of progressive progression, also cause a dramatic increase in chronic renal failure.
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