Uric acid nephrosis refers to because uric acid produces overmuch or excrete reduce, cause uric acid to cause kidney damage. There are two types of kidney damage: urinary calculi and uric acid parenchymal damage.
There are three kinds of clinical manifestation:
Chronic hyperuricemia: more common in middle-aged men. More often accompanied by gouty arthritis or gout stone; kidney damage early manifestation of mild proteinuria, a small amount of red blood cells and urine concentration function decline; late stage of hypertension, renal dysfunction, a small number lead to uremia. The kidney is alive and thrifty, showing the presence of radiation like needle shaped uric acid crystals and chronic inflammatory changes in the renal interstitium.
Acute uric acid nephropathy: the onset of acute, more common in myeloproliferative diseases and malignant tumors, radiotherapy, chemotherapy. The clinical manifestations include oliguria or oliguria, and rapid deterioration of renal function (acute renal failure). Large amounts of uric acid crystals and red blood cells are visible in urine. If not treated in time, the condition continues to worsen and the patient eventually dies of renal failure.
Uric acid stones: more asymptomatic, can occur hematuria, accompanied by or without renal colic, stone for light transmission, abdominal plain film more than can not be displayed, often through intravenous pyelography to be found. Urolithic composition for uric acid or its salts.
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