Hypertensive nephropathy check items

Hypertensive nephropathy check items

1, history and symptoms

More than 40 to 50 years of age, high blood pressure history of 5 to 10 years. Early only nocturia increased, followed by proteinuria, inpidual cases may be due to capillary rupture and transient ocular hematuria, but not with significant low back pain. Often associated with atherosclerotic retinopathy, left ventricular hypertrophy, coronary heart disease, heart failure, cerebral arteriosclerosis and / or cerebrovascular accident history.

2, physical examination found

Generalized blood pressure continued to increase (20.0 / 13Kpa, 150 / 100mmHg above); some eyelids and / or lower extremity edema, heart sector expansion; most arteriosclerotic retinopathy, when the eye has a stripe, flame-like bleeding and cotton Soft exudation, support for the diagnosis of malignant renal arteriosclerosis. Accompanied by hypertensive encephalopathy may have the corresponding signs of the nervous system.

3, auxiliary inspection

(Red blood cells, white blood cells, transparent tube type) less, may have hematuria; early increase in serum uric acid, urinary NAG enzyme, β2-2g, MG increased, urine concentration - dilution dysfunction; Ccr more slowly decreased, blood urea nitrogen, creatinine increased. Renal tubular damage more than glomerular damage.

4, check

Generalized blood pressure continued to increase (20.0 / 13Kpa, 150 / 100mmHg above); some eyelids and / or lower extremity edema, heart sector expansion; most arteriosclerotic retinopathy, when the eye has a stripe, flame-like bleeding and cotton Soft exudation, support for the diagnosis of malignant renal arteriosclerosis. Accompanied by hypertensive encephalopathy may have the corresponding signs of the nervous system.

5, check

Mostly mild to moderate proteinuria, 24 hours more quantitative in 1.5 ~ 2.0g. (Erythrocyte, white blood cells, transparent tube type), may have hematuria; early increase in serum uric acid, urinary NAG enzyme, β2-MG increased urine thickening - dilution dysfunction; Ccr more slowly decreased, blood urea nitrogen , Creatinine increased. Renal tubular damage more than glomerular damage.

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