CRF patients due to the internal environment disorders and immune function decreased, susceptible to in vitro and in vivo factors such as the impact of pulmonary lesions, mainly uremia lung, pulmonary edema, pleural effusion and so on.
Uremia lung: also known as uremia pulmonary edema, uremia pneumonia. The symptoms are mild, early uremia only caused by systemic symptoms, with the development of the disease gradually mild to moderate cough, cough a small amount of sticky sputum and difficulty breathing. Development of interstitial fibrosis, dyspnea and cyanosis increased. Small and medium amount of hemoptysis is also an important symptom. And cardiogenic pulmonary edema, pulmonary infection and pulmonary hemorrhage, nephritis syndrome phase identification.
Uremic pleural disease: the incidence of 15% to 20%, pleural friction, chest pain or chest discomfort, difficulty breathing or fever. Pleural friction lasted 1 to 15 days, may be associated with exudation. There was no relationship between blood urea nitrogen and exudation.
Pulmonary calcification: CRF often causes soft tissue calcification, lung is the most common site. The clinical manifestations of chronic dyspnea or acute, subacute respiratory failure, chest radiograph can be completely normal. Stop calcium, resection of parathyroid, low-phosphorus diet, oral aluminum oxide and the application of low calcium dialysate, increase the number of dialysis or duration can reverse the calcification.
Uremic pulmonary edema: one of the common acute diseases of the Department of Nephrology. When CRF patients, especially with oliguria, such as sudden severe breathing difficulties, sit breathing, with fear, suffocation, looking green, lips cyanosis, sweating, cough, sputum, may be associated with hemoptysis and a large number of pink foam sputum, lung symmetry covered with wet rales and wheeze, heart rate, pulse thin should be considered as acute pulmonary edema.
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